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(PubMed) Cooper JM, Korlipara LV, Hart PE, Bradley JL, Schapira AH

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This includes developing bifunctional molecules (e.g., Fer-1 conjugated to Nec-1, a necroptosis inhibitor) that co-target complementary pathways or epigenetic editing tools (e.g., dCas9-SunTag-KRAB systems) to dynamically suppress the transcription of ferroptosisnecroptosis crosstalk nodes (e.g., RIPK3)

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