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NSAIDs are a common cause of gastric ulcers and intestinal lesions due to their inhibition of prostaglandin synthesis, which is crucial for mucosal defense

Although melanoma cells do not produce IGF-1, activation of the MAPK and PI3K signaling pathway by paracrine stimulation of IGF-1 from stromal fibroblasts enhanced survival, migration, and growth of melanoma cells only from biologically early tumors (47)

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