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[25] With increasing numbers of proliferating stem cells in the germ line, NAD+ repletion greatly prolonged lifespan and postponed the onset of accelerated aging in animal models

In other words: if a researcher is studying IGF-1 LR3's effects on cell proliferation and the preparation is contaminated with endotoxin, the observed proliferation response could be driven by LPS rather than by IGF-1 LR3 signaling through IGF-1R

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doi: 10.1016/j.ijpharm.2025.125318 24

Which raised the question, if the levels of glutathione were restored in cells, would the mitochondria be recharged and able to provide power to the cell