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This, in turn, results in impaired dehydrogenase activity and a decrease in TCA flux (Svobodov et al
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Material and methods: The review authors independently screened and evaluated studies identified through medical databases, including PubMed, Scopus, and institutional repositories
However, decreased responsiveness to VIM DBS by select body parts (possibly due to the somatotopic organization of the VIM nucleus) increased tolerance to VIM stimulation due to compensation or disease progression [176, 177], and the development of adverse side effects, such as dysarthia and dysphagia [174], in patients, has driven efforts to optimize DBS targeting
