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They further found that activation of the Wnt/-catenin signaling pathway in human airway epithelial cells from COPD smoking patients resulted in increased Vimentin expression, increased fibronectin release, and enhanced TGF-1/Smad signaling

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Interestingly, other CYP11B-derived metabolites, such as 11-hydroxyandrostenedione (11-OHA4), did not follow the same trend, suggesting that the increase in 11-OHP4 is metabolite-specific and not due to generalized upregulation of CYP11B enzymes

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