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This same study found that cerebral vasculature experiences prolonged up-regulation of MHCI and MHCII, following systemic bacterial infection (Puntener et al., 2012), suggesting cells of the vasculature also remain in a primed state

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PMCID: PMC9556195

Furthermore, EA attenuated pathological damage and neuronal apoptosis in rats with TBI, leading to gradual improvement in pathological morphology, such as nuclear consolidation, fragmentation, cell swelling, neuronal necrosis, and scar tissue

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