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Perfusion deficits can accelerate clinical deterioration and are associated and correlated with adverse treatment outcomes (Horgusluoglu et al

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Anti-inflammatory M2, but not pro-inflammatory M1 macrophages promote angiogenesis in vivo

[61,63] have reported that the small molecule antidepressant drug amitriptyline is capable of binding to the BDNF target Trk receptor, induce receptor dimerization, and autophosphorylation