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A pharmacokinetic study was conducted in rats following oral administration of mildronate at doses of 40160 mg/kg

M107 and its broad metal selectivity

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Further investigation revealed that the IC50 concentration of CGA, through the depletion of free iron within hepatocytes, significantly reduced the expression of iron-sulfur cluster subunits in mitochondrial complexes I-III and disrupted the oxidative-reductive homeostasis of hepatocytes, ultimately leading to hepatotoxicity

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