10.4103/ijmr.IJMR_2065_17 13 BlackwellT
Protocol registration The protocol of this study was not registered
When produced excessively, it may suppress the release of Thyroid Stimulating Hormone from the pituitary. 18 It also forces T4 to be converted to reverse T3 (rT3) which actually blocks thyroid function, even though thyroxine levels look adequate when tested. 19 Blood glucose imbalance also triggers cortisol secretion, and insulin resistance may have a direct negative effect on the thyroid, too
Additionally, the levels of triglyceride and cholesterol were reported significantly decreased compared to the placebo group
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To further elucidate the prognostic relevance of GSTA4 in PC, survival analyses were performed across multiple cohorts of patients with PC, including disease-free survival, disease-specific survival, overall survival, progression-free survival, and relapse-free survival
