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Other postmortem surveys of three COVID-19 patients with severe symptoms showed that the cerebral cortex had increased numbers of reactive astrocytes and exacerbated microglial activation, as well as decreased glutathione levels and up-regulation of genes related to IL-1B, IL-6, interferon-inducible transmembrane, MX dynamin-like GTPase 1, and 2,5-oligoadenylate synthetase 2, suggesting a relationship between oxidative stress and neuroinflammation mediated by glia (Boroujeni et al
Bee venom melittin disintegrates the respiration of mitochondria in healthy cells and lymphoblasts, and induces the formation of non-bilayer structures in model inner mitochondrial membranes
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