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The activation of caspases in response to oxidative stress was closely linked to the observed reduction in aminophospholipid translocase activity and the increased PS externalization in aged RBCs

At the physiological phenotype level, CRISPRa has also been used to increase the transcription of obesity risk genes SIM1 and MC4R , and to successfully rescue haploinsufficient obesity in mouse modelswith the effects persisting up to 9 months post-treatment [80]

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