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PubMed, Chappell, Kenneth, et al

Its ability to preserve mitochondrial integrity, enhance cerebral perfusion, mitigate oxidative stress, and support synaptic plasticity positions it as a neuroprotective adjunct rather than a primary monoaminergic antidepressant

Mechanistic Interests Matrix signaling, growth-factor pathway modulation, inflammatory signaling, nitric oxide pathway interactions

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Some of these may be borne out as the trials read out, and the fair summary for now is that promotion has run ahead of the human evidence

Alterations of oligodendrocyte and myelin energy metabolism in multiple sclerosis