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Cancer cells exploit this paradox by modulating their redox state to support survival, metastasis, and therapeutic resistance

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ISSN 1036-7314

Methods An in vitro model of PCOS was constructed by treating KGN cells with dihydrotestosterone (DHT), and PLB treatment, YTHDF1 knockdown (si-YTHDF1), and SLC7A5 overexpression (pcDNA 3.1-SLC7A5) were intervened respectively

Soluble protein, MDA, H 2 O 2 , and total antioxidant capacity Soluble protein was quantified by the Coomassie Brilliant Blue G-250 assay

doi:10.1016/j.nbd.2012.09.015 3