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The nephrotoxic mechanisms of pCS primarily involve inducing oxidative stress through activation of nicotinamide adenine dinucleotide phosphate (NADPH) oxidase, triggering inflammatory responses, and promoting renal fibrosis (124)

Acknowledgements This paper is in memory of Professor Adrienne Gordon, the original AG, who advised us on this project and many past ones

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Plasmid construction All plasmids were constructed using either restriction enzyme cloning or Gibson assembly

The individuals included in the analysis represented both the impaired and non-impaired groups

10 The STAT3/STAT1 cascade can be increased in keratinocytes through regulation of factors that sense both oxidative stress and reduced levels of oxygen