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Furthermore, intracellular Fe 2+ is pumped into the extracellular space through membrane ferritin 1 (SLC40A1/FPN1) to strictly maintain cellular iron homeostasis ( Many studies have demonstrated that iron metabolism-related proteins could be used as targets to regulate ferroptosis

Addressing the key scientific questions discussed in this review will improve our understanding of the precise role of ferroptosis in various retinal diseases, thereby providing a new scientific basis for the prevention and treatment of retinal diseases with ferroptosis-targeted drugs

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One promising direction is to repurpose metabolic cues as both assembly triggers and cellular pathway regulators, enabling bidirectional communication between supramolecular nanomaterials and cellular responses