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glutathione liver damage

glutathione liver damage disulfide sensitizes hepatocytes to TNFα-mediated cytotoxicity via IKK-β S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease A near-infrared fluorescent probe based

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Description

2017 Oct 1;186:66-79

glutathione liver damage disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease A near-infrared fluorescent probe based

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glutathione liver damage disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease A near-infrared fluorescent probe based

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glutathione liver damage disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease A near-infrared fluorescent probe based

Serum high-sensitivity C-reactive protein, tumor necrosis factor-, interleukin (IL)-1, IL-17A and IL-23 levels in patients with hidradenitis suppurativa

glutathione liver damage disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease A near-infrared fluorescent probe based

doi: 10.1016/j.mehy.2020.109862 220 PoljsakB.uputD.MilisavI

glutathione liver damage disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease A near-infrared fluorescent probe based

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glutathione liver damage disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease A near-infrared fluorescent probe based
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