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Mice deficient in the Shmt2 gene have mitochondrial respiration defects and are embryonic lethal

Zhiqi Hu provided many suggestions during study design and manuscript preparation

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doi: 10.1089/ast.2006.6.463, 42 HanR.ZhangX.LiuJ.LongQ.ChenL.LiuD.et al

However, direct experimental evidence supporting this hypothesis in ovarian models is currently lacking, and future studies will be required to establish whether MAMs-mediated phospholipid remodeling plays a causal role in these processes

In summary, this study showed that although most circulating IGF-1 is derived from hepatocytes, it is not the main IGF-1 that is required for postnatal growth and development (303)