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In hepsin-deficient (hepsin / ) mice, a notable inhibition of HGF-cMet signaling was observed, characterized by decreased levels of activated HGF and reduced cellular abundance of phosphorylated/activated cMet

These combined effects make them particularly valuable for studies focused on injury recovery, inflammation, and tissue stability

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Laboratory studies have demonstrated biological activity at concentrations as low as 0.01 nanomolar, showing measurable effects on fibroblast activity and collagen production even at these minute levels

10.1038/s44220-023-00145-6 232 KelmendiB.AdamsT